Head & Neck Anatomy · Ophthalmology

Extraocular Muscles

Six extraocular muscles move the eye, supplied by three cranial nerves. One rule summarises the innervation: LR6, SO4, all others 3.

✦ The Mnemonic

"LR Six, Superior Four, All Others Third"

CN VI → Lateral Rectus · CN IV → Superior Oblique · CN III → all four remaining muscles

SR Superior Rectus (CN III) Primary: elevation; secondary: intorsion, adduction
IR Inferior Rectus (CN III) Primary: depression; secondary: extorsion, adduction
MR Medial Rectus (CN III) Pure adduction
LR Lateral Rectus (CN VI) Pure abduction
SO Superior Oblique (CN IV) Primary: intorsion; secondary: depression, abduction
IO Inferior Oblique (CN III) Primary: extorsion; secondary: elevation, abduction

📚 Clinical Breakdown

CN III palsy: eye 'down and out' (unopposed SO + LR), ptosis. If parasympathetic fibres compressed: fixed dilated pupil. CN IV palsy: most common cause of vertical diplopia; patients tilt head away from the affected side to compensate for extorsion. CN VI palsy: convergent squint, esotropia, horizontal diplopia.

The superior oblique passes through the trochlea (fibrocartilaginous pulley, medial orbital wall) before inserting on the posterior-superior globe. CN IV has the longest intracranial course — most vulnerable in closed head injury.

Internuclear ophthalmoplegia (INO): MLF lesion disconnects CN VI nucleus from the contralateral CN III nucleus. On horizontal gaze, the ipsilateral eye fails to adduct; contralateral eye shows nystagmus. Bilateral INO in a young adult = MS until proven otherwise.

LR nerve CN VI (abducens)
SO nerve CN IV (trochlear)
CN III palsy Down and out + ptosis ± dilated pupil
INO cause MLF lesion; bilateral = MS

⭐ Clinical Pearl

Bilateral INO in a young adult is essentially pathognomonic of multiple sclerosis. The MLF connects the ipsilateral CN VI nucleus to the contralateral CN III nucleus — a lesion here produces adduction failure on the ipsilateral side with contralateral abducting nystagmus.

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