Causes of Anaemia
Anaemia (Hb <130 g/L men, <120 g/L women) is classified by MCV into microcytic, normocytic, and macrocytic. Each size category has a distinct differential diagnosis.
✦ The Mnemonic
"TILSA Finds Microcytic; BLAND Macrocytic"
Microcytic (MCV <80): TILSA · Macrocytic (MCV >100): BLAND
Clinical Breakdown
Iron studies interpretation: Iron deficiency = low serum iron, low ferritin, raised TIBC. Anaemia of chronic disease = low serum iron, raised ferritin (acute phase protein), low TIBC. The key distinction: ferritin is high in ACD and low in IDA — ferritin is the single most useful test.
B12 deficiency vs folate deficiency: both cause megaloblastic macrocytic anaemia with hypersegmented neutrophils. B12 deficiency also causes subacute combined degeneration of the spinal cord (dorsal and lateral columns) — folate does not. Always check B12 before treating with folate (giving folate to a B12-deficient patient can precipitate cord degeneration).
Haemolytic anaemia is normocytic: raised reticulocytes (haemopoietic response), raised bilirubin (unconjugated), raised LDH, low haptoglobin, raised urobilinogen. Causes: immune (warm or cold AIHA), hereditary (spherocytosis, elliptocytosis, G6PD, sickle cell), microangiopathic (TTP, HUS, DIC), mechanical (prosthetic valve).
⭐ Clinical Pearl
Sickle cell disease: HbS polymerises when deoxygenated, causing sickling and vaso-occlusion. The sickling crisis — acute painful crisis — is triggered by: cold, dehydration, infection, hypoxia. Management: analgesia (opioids), hydration, oxygen, antibiotics if infection. Annual transcranial Doppler screening identifies children at stroke risk (prophylactic exchange transfusion).